If it doesn't grow, swim, walk, or fly, or if it comes with an ingredient list, I don't eat it. Meat, seafood, eggs, and whole vegetables and fruit are the entire menu. No packaged snacks, no bars, no cereal, no bread, no seed oils, no artificial sweeteners, no "low-carb" processed substitutes pretending to be food.
This isn't a weight-loss diet with an end date โ it's a permanent, prevention-first eating pattern built around one question: does this food give my body real information, or does it confuse it? Whole, unprocessed food gives clean signals. Processed food โ even "healthy" processed food โ is noise, and noise is what drives insulin resistance, inflammation, and gut dysfunction over decades.
A 16:8 eating window, roughly 12pm to 8pm. I stop eating by 8pm and don't eat again until around noon the next day โ an overnight fast of about 16 hours, with all meals falling inside the remaining 8-hour window. Water and electrolytes without sugar are fine during the fasted stretch; food isn't.
This isn't a separate program bolted onto the food rules above โ it's the timing half of the same approach. A consistent multi-hour daily fast gives insulin a real chance to come back down between meals, which matters directly for the triglyceride and small-dense-LDL work already happening through the food choices themselves. It also lines up with the rest of the daily structure: the first supplements are taken fasted at 7:30am, and the eating window opens with the first meal around 11amโ12pm.
Sugar and refined carbs drive the exact metabolic problems I'm working to reverse โ elevated triglycerides, small dense LDL particles, insulin resistance. Removing them at the source does more than any supplement can. Whole fruit and vegetables still provide carbohydrate, fiber, and micronutrients โ just without the insulin spike of refined starch or added sugar riding along with it.
"Processed" is its own separate problem, independent of carbs. Emulsifiers and additives common in packaged food โ even sugar-free ones โ measurably disrupt the gut microbiome, including the exact keystone bacterial species (like F. prausnitzii and Akkermansia) I'm actively working to rebuild. A "zero-sugar" protein bar can still be off-limits if the ingredient list is long and synthetic โ the additive burden matters as much as the sugar content.
Protein and nutrient density come first. This isn't restriction for its own sake โ the goal is to make every calorie count toward muscle, micronutrients, and metabolic health, not to eat as little as possible.
This way of eating isn't just a general preference โ it's currently load-bearing for an active project: rebuilding two keystone gut bacteria, Akkermansia muciniphila and Faecalibacterium prausnitzii, that showed undetectable on both prior GI-MAP draws. That rebuild depends on diet at least as much as it depends on the supplements (Akkermansia 500 PRO, Sunfiber PHGG, Tri-Butyrin, MegaPre) โ so eating whole-foods-only isn't a nice-to-have alongside the protocol, it's part of the protocol.
Whole food does double duty here: cutting out additives removes the headwind, while specific whole foods actively feed the rebuild โ
| Target species | Whole foods that help |
|---|---|
| Akkermansia (polyphenols) | Unsweetened cranberries, EVOO, whole grapes |
| F. prausnitzii (resistant starch) | Cooked-and-cooled potato/rice, green banana, avocado, asparagus |
| Gut lining / diversity | Bone broth, unpasteurized fermented vegetables (no additives) |
There's also a retest riding on this: the plan is to stop the bridge supplements (Tri-Butyrin, MegaPre) 6โ8 weeks before the January 2027 GI-MAP retest, so the retest reflects whether Akkermansia and Sunfiber โ plus diet โ have durably reestablished these species, not whether the bridge agents are still propping things up in the moment. A diet slip doesn't just risk a bad week; it risks muddying that readout.
This is the part that separates this way of eating from a diet I picked because it sounded good: the specific inclusions and exclusions above aren't guesses. They're derived from my own genetic testing and bloodwork, run through a real decision process, the same way a clinician would use a patient's chart. Two data sources dictate the food rules directly.
23andMe testing surfaced variants in three genes that directly changed what counts as "food" versus "avoid" for me, even within categories that are otherwise considered healthy:
| Gene variant | What it means | Food rule it creates |
|---|---|---|
| MTRR | Impaired ability to activate synthetic B12 | Cyanocobalamin (the synthetic B12 in fortified cereal, cheap protein shakes, energy drinks) is out. Real food or hydroxycobalamin/methylcobalamin only. |
| MTHFR A1298C | Reduced conversion of synthetic folic acid to usable folate | "Enriched/fortified" folic-acid products (flour, cereal, most protein powders) are out. Folate has to come from real food โ leafy greens, legumes, citrus, beets, asparagus. |
| COMT (homozygous) | Slower clearance of catechols, including concentrated EGCG | Green tea extract/supplement products are out โ concentrated EGCG is the thing to avoid, so it stays off the list entirely. |
| APOE ฮต4 | Higher sensitivity of cardiovascular risk to fat quality | Fat sourcing leans deliberately toward monounsaturated (EVOO, avocado) over saturated-heavy โ this variant is exactly why fat quality, not just "low-carb = eat more fat," is the operating rule. |
Bloodwork functions as an ongoing report card on whether the diet is working, and specific markers have directly shaped specific choices:
| Marker / finding | What it drove |
|---|---|
| Cholesterol, ApoB & LDL particles | Total cholesterol (283โ171) and LDL-C (212โ111) came down without a statin (Feb 2024 โ Sep 2026), alongside cutting refined carbs/sugar โ a major driver of small, dense atherogenic particles โ rather than cutting dietary fat broadly. Latest panel (9/14/2026): total 171 ยท LDL-C 111 ยท HDL-C 47 ยท non-HDL 124 mg/dL (the LDL-C here is calculated differently from earlier Function Health values). ApoB fell from 139 to 93 mg/dL (Jan 2025 โ Jul 2026) but is not part of that basic panel, and LDL particle count was still elevated in July (1,453 nmol/L) โ so this is working, not finished. |
| Triglycerides | Most directly tied to added sugar and refined carbohydrate intake of any lipid marker โ the single biggest reason "no added sugar" is a hard line rather than a preference. Triglycerides went from 162 (Feb 2024) to 66 mg/dL on 9/14/2026, the lowest on record. Two July readings (144 and 153) were drawn during a supplement washout and did not hold on retest. |
| Uric acid | Kept as a watch flag โ purine-dense foods (organ meats, shellfish, anchovies/sardines in quantity) aren't banned, but portioned with this marker in mind rather than eaten freely. |
| GI-MAP (Akkermansia, F. prausnitzii undetectable) | See the Phase 4 section above โ this is the clearest example of a lab result directly rewriting what's on the plate. |
The through-line: every food rule on this page can be traced back to either a whole-foods-first principle, a specific gene variant, or a specific lab marker. Nothing here is arbitrary, and nothing here is permanent dogma โ if the labs and the genetics say something different one day, the diet moves with the data, not the other way around.
People sometimes ask what "diet" this is โ it doesn't map cleanly onto one label, but borrows the best-evidenced piece of a few: